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City Ann Arbor / Oxford / Oberkochen / Fremont / Chongqing / St. Louis / Santa Cruz / / Company Jackson Laboratory / Sigma-Aldrich / Cayman / Eisai Inc. / Macmillan Publishers Limited / / Country Germany / United States / United Kingdom / China / / / Event Product Issues / / Facility Bar Harbor / Jackson Laboratory / Daping Hospital / Third Military Medical University / Institute of Surgery Research / Laboratory Animals / / / IndustryTerm food / Online Publication / / MedicalCondition inflammatory reaction / inflammatory bowel disease / intracellular lipid disorder / TLR4-mediated inflammatory cytokine expression / ACAT1 deficiency VSMCs / inflammatory properties / Disease mice / oxLDL-induced inflammation / intracellular inflammation / inflammatory cytokines / disorder / TLR4/MyD88/NF-κB inflammatory signaling / cardiovascular diseases / inflammatory genes / ACAT1 deficiency / TLR4 deficiency / Disease / Atherosclerosis / knockout-mediated gene deficiency / secondary necrosis / TLR4-mediated inflammation / TLR4mediated inflammation / TLR4/ MyD88/NF-κB inflammatory signaling / tumor necrosis / myocardial infarction / B inflammatory signaling pathway / TLR4-mediated inflammatory mechanism / diseases / advanced atherosclerosis / inflammation / lipid metabolism disorder / / Organization Institute of Surgery Research / Animal Research Committee / Third Military Medical University / Department of Neurology / US Federal Reserve / Daping Hospital / / / Position representative / / Product TLR4 / Rosiglitazone / foam cell formation / atherosclerotic plaque formation / VSMC foam cell formation / / ProvinceOrState South Dakota / Yuzhong District / / Technology Antibodies / siRNA / Biotechnology / apoptosis / gene expression / / URL www.nature.com/cddis / / SocialTag 
TLR4-mediated inflammation promotes foam cell formation of vascular smooth muscle cell by upregulating ACAT1 expression