An upper limit for macromolecular crowding effects

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Apoptosis Immune system Proteins Signal transduction Tumor necrosis factor-alpha Fas receptor Apoptosome Insulin Caspase 3 Biology Cell biology Programmed cell death		An upper limit for macromolecular crowding effects Add to Reading List Source URL: www.ncbi.nlm.nih.gov Download Document from Source Website File Size: 448,10 KB Share Document on Facebook

	Original Research Article Cuscuta campestris induces apoptosis by increasing reactive oxygen species generation in human leukemic cells Maliheh Moradzadeh 1, Azar Hosseini 2, Hasan Rakhshandeh 2, Azita Aghaei 2, Hamid R DocID: 1vjZS - View Document
	Boswellic acid acetate induces apoptosis through caspase-mediated pathways in myeloid leukemia cells Lijuan Xia, Duo Chen, Rui Han, et al. Mol Cancer Ther 2005;4:Updated version DocID: 1v6bB - View Document
	Arachidonic Acid Anomalously Accumulates after Archetypic Apoptosis at Aardvark Association Areas. Anna Author, Aaron Associate, and Alana AdvisorDept. of Neuroscience, Univ. of Affiliation Medical School, Affil DocID: 1uqPT - View Document
	Primed to Perish: Heightened Mitochondrial Priming Explains hESC Apoptosis Sensitivity DocID: 1tZtH - View Document
	Cellular Signalling – 725 The somatostatin analogue TT-232 induces apoptosis in A431 cells Sustained activation of stress-activated kinases and inhibition of signalling to extracellular signal-regulated DocID: 1tOA0 - View Document