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FANCD2 / Formaldehyde / H2AFX / Ataxia telangiectasia mutated / FANCL / Dox / Homologous recombination / Biology / DNA repair / Fanconi anemia


B R I E F C O M M U N I C AT I O N S Formaldehyde catabolism is essential in cells deficient for the Fanconi anemia DNA-repair pathway Metabolism is predicted to generate formaldehyde, a toxic,
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Document Date: 2014-06-02 11:49:23


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City

Cambridge / Trewick / /

Company

IgG / MRC Laboratory / Sigma-Aldrich / Nature America Inc. / Children’s Leukaemia Trust / I O N S a / 1Medical Research Council Laboratory / /

Country

Japan / United Kingdom / /

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Event

Product Issues / /

Facility

Kihara Institute / MRC Laboratory of Molecular Biology / Addenbrooke’s Hospital / Kyoto University / Research Council Laboratory of Molecular Biology / Radiation Biology Center / /

IndustryTerm

less reactive and more stable product / gene products / ONLINE PUBLICATION / thiol / chemical insight / /

MedicalCondition

Fanconi anemia / /

Organization

Kihara Institute for Biological Research / Department of Medicine / Fanconi Anaemia Research Fund / Radiation Biology Center / Addenbrooke’s Hospital / Department of Late Effect Studies / Kyoto University / Kyoto / /

Person

Rosado / Nat / Crossan / /

Position

AUTHOR / G.P. / Fisher / /

Product

doxycycline / cell lines / /

ProvinceOrState

South Carolina / /

PublishedMedium

8 Days / Molecular Biology / /

Technology

Radiation / stem cells / recombination / /

URL

http /

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